Cystic fibrosis transmembrane conductance regulator (CFTR) may be the principal apical route for transepithelial fluid transport induced by enterotoxin. (EC), (-)Cepigallocatechin (EGC), (-)Cepicatechin-3-gallate (ECG) ITSN2 and EGCG revealed that ECG also experienced CFTR inhibitory activity. EGCG dose-dependently and reversibly inhibited CFTR Cl- route 3-deazaneplanocin A HCl IC50 activity in transfected FRT cells with an IC50… Continue reading Cystic fibrosis transmembrane conductance regulator (CFTR) may be the principal apical